Trauma's Lethal Diamond: Coagulopathy, Acidosis, Hypothermia and Hypocalcemia

Author(s): Justin J Milici, MSN, RN, CCRN, CEN, CPEN, TCRN, FAEN

Contact Hours 1.00

CERP A 1.00

Expires Dec 31, 2027

Topics: Hematology/Oncology, Trauma

Population: Adult

Role: Staff

Fees
Member: Free
NonMember: $10.00

Added to Collection

Activity Summary

Early recognition and aggressive management of coagulopathy, hypothermia, and acidosis are key to increasing survival and optimizing outcomes for the injured patient. Recent data also show that hypocalcemia can exacerbate the lethal effects of the trauma triad. This presentation examines how calcium plays a significant role in trauma resuscitation and the deleterious effects of hypocalcemia.

Objectives

  • Discuss the damaging effects of coagulopathy, hypothermia and acidosis in the injured patient.
  • Summarize the physiologic role of calcium and the effects of hypocalcemia in coagulopathy, hypothermia and acidosis.
  • Explain the complications of coagulopathy, hypothermia, acidosis and hypocalcemia and steps to mitigate them.

Continuing Education Disclosure Statement

Criteria for Awarding Contact Hours

Learners must complete the entire activity and the associated evaluation AND read Implicit Bias impacts patient outcomes. No partial credit will be awarded.

Accreditation

American Association of Critical-Care Nurses is accredited as a provider of nursing continuing professional development by the American Nurses Credentialing Center's Commission on Accreditation.
Provider approved by the California Board of Registered Nursing, Provider number CEP 1036, for {contactHours} contact hours.

Disclosures

The Nurse Planner has determined that no individuals with the ability to control content of this activity have relevant relationships with ineligible companies.

Activities with pharmacotherapeutic credit are to assist the APRN in fulfilling their education requirements for licensure and certification renewals.

Refund Policy

Continuing Education Activities are nonrefundable.